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By using our content, services, or application you declare to be bound by the License Agreement.. Tools for activation/deactivation: 9.3, 9.2, 9.2.1, 9.1, 9, 8.3, 8.2, 8.1.Manifestation of the intestinal siderophore receptor FepA in Escherichia coli increases the virulence potential of avian pathogenic Escherichia coli and promotes survival in iron-rich conditions.
The enterobacterial adherence factor FepA is known as a major target of bovine serum albumin in that it mediates bacterial attachment to bovine brush border membranes and promotes bacterial invasion into cultured eukaryotic cells. It also increases the adhesion of Salmonella enterica serovar Typhimurium to chicken intestinal epithelial cells. The objective of this study was to characterize the contribution of the fepA gene to the colonization ability of avian pathogenic Escherichia coli (APEC) in chickens. On the basis of the fepA genotyping, fepA-positive strains were found to be associated with high levels of serum resistance and high adherence potential to Caco-2 cells and HEp-2 cells. The fepA-deficient mutant strain was attenuated in virulence in chickens. However, the mutant strain was re-isolated from the spleen and liver after the chickens were killed. A comparison of bacterial growth in the presence of FeCl(3) (iron is known to enhance FepA expression) or FeCl(2) demonstrated that the bacterium grew better in the presence of FeCl(3). Bacteria attached to HEp-2 cells or trachea tissues